Proinflammatory synergism of ethanol and HIV-1 Tat protein in brain tissue.

Dietary fat interacts with PCBs to induce changes in lipid metabolism in mice deficient in low-density lipoprotein receptor.

Signaling mechanisms of HIV-1 Tat-induced alterations of claudin-5 expression in brain endothelial cells.

Signaling mechanisms of HIV-1 Tat-induced alterations of claudin-5 expression in brain endothelial cells.

[Anonymous].  2005.  Signaling mechanisms of HIV-1 Tat-induced alterations of claudin-5 expression in brain endothelial cells.. Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism. 25(9):1159-70.

HIV-1 Tat protein-induced alterations of ZO-1 expression are mediated by redox-regulated ERK 1/2 activation.

HIV-1 Tat protein-induced alterations of ZO-1 expression are mediated by redox-regulated ERK 1/2 activation.

[Anonymous].  2005.  HIV-1 Tat protein-induced alterations of ZO-1 expression are mediated by redox-regulated ERK 1/2 activation.. Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism. 25(10):1325-35.

HIV-Tat protein induces P-glycoprotein expression in brain microvascular endothelial cells.

Mechanisms of the blood-brain barrier disruption in HIV-1 infection.

Modification of environmental toxicity by nutrients: implications in atherosclerosis.

Zinc deficiency increases plasma lipids and atherosclerotic markers in LDL-receptor-deficient mice.

HIV-TAT protein upregulates expression of multidrug resistance protein 1 in the blood-brain barrier.

HIV-TAT protein upregulates expression of multidrug resistance protein 1 in the blood-brain barrier.

[Anonymous].  2006.  HIV-TAT protein upregulates expression of multidrug resistance protein 1 in the blood-brain barrier.. Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism. 26(8):1052-65.

Linoleic acid induces proinflammatory events in vascular endothelial cells via activation of PI3K/Akt and ERK1/2 signaling.