Delta-9-tetrahydrocannabinol-dependent alterations in brain structure.

Modulation of brain aging correlates by long-term alterations of adrenal steroids and neurally-active peptides.

Redistribution of synaptic vesicles during long-term potentiation in the hippocampus.

Probable Ca2+-mediated inactivation of Ca2+ currents in mammalian brain neurons.

'Increased calcium-current' hypothesis of brain aging.

Postsynaptic membrane shifts during frequency potentiation of the hippocampal EPSP.

Commentary on biomarkers of aging in the nervous system.

Quantitative changes in hippocampal structure following long-term exposure to delta 9-tetrahydrocannabinol: possible mediation by glucocorticoid systems.

Quantitative changes in hippocampal structure following long-term exposure to delta 9-tetrahydrocannabinol: possible mediation by glucocorticoid systems.

[Anonymous].  1988.  Quantitative changes in hippocampal structure following long-term exposure to delta 9-tetrahydrocannabinol: possible mediation by glucocorticoid systems.. Brain research. 443(1-2):47-62.

Synaptic vesicle redistribution during hippocampal frequency potentiation and depression in young and aged rats.

Hippocampal neurobiological mechanisms of age-related memory dysfunction.