Vitamin E attenuates induction of elastase-like activity by tumor necrosis factor-alpha, cholestan-3 beta,5 alpha,6 beta-triol and linoleic acid in cultured endothelial cells.

Vitamin E attenuates induction of elastase-like activity by tumor necrosis factor-alpha, cholestan-3 beta,5 alpha,6 beta-triol and linoleic acid in cultured endothelial cells.

[Anonymous].  1993.  Vitamin E attenuates induction of elastase-like activity by tumor necrosis factor-alpha, cholestan-3 beta,5 alpha,6 beta-triol and linoleic acid in cultured endothelial cells.. Clinica chimica acta; international journal of clinical chemistry. 215(2):201-11.

Selective disruption of endothelial barrier function in culture by pure fatty acids and fatty acids derived from animal and plant fats.

Selective disruption of endothelial barrier function in culture by pure fatty acids and fatty acids derived from animal and plant fats.

[Anonymous].  1993.  Selective disruption of endothelial barrier function in culture by pure fatty acids and fatty acids derived from animal and plant fats.. The Journal of nutrition. 123(7):1208-16.

Proteoglycans and endothelial barrier function: effect of linoleic acid exposure to porcine pulmonary artery endothelial cells.

Nutrition, endothelial cell metabolism, and atherosclerosis.

Fatty acid-mediated effects on the glutathione redox cycle in cultured endothelial cells.

Disruption of endothelial barrier function: relationship to fluidity of membrane extracellular lamella.

Influence of nutrients and cytokines on endothelial cell metabolism.

Zinc and endothelial function.

Tumor necrosis factor reduces proteoglycan synthesis in cultured endothelial cells.

Electron spin resonance studies of fatty acid-induced alterations in membrane fluidity in cultured endothelial cells.